T or F: If You Get Non-Melanoma Skin Cancer…?
Question by health_avenger: T or F: If you get non-melanoma skin cancer…?
…(e.g. skin cancer associated with cumulative sun exposure) you may have a greatly reduced risk of breast, colon, prostate and other solid cancers.
Hints:
http://www.mercola.com/2004/feb/11/vitamin_d_types.htm
http://www.bmj.com/cgi/content/extract/334/7607/1295-d
http://cebp.aacrjournals.org/cgi/content/abstract/16/3/422
Best answer:
Answer by Alt Healer
Hi Health Avenger
I see your interested in Health, but not sure what the question is here. Ill post some info on skin cancer Im aware of it. This is very powerful.
Research and Facts about the Dangers of Sunscreen
Being concerned about skin cancer is valid; however, most sunscreen products can do more harm than good. Some sunscreen products contain toxic chemicals that penetrate the skin, and can actually increase the risk of disease.
The following are a few highlighted remarks and findings by numerous experts whose research shows that many ingredients in sunscreen are simply not safe and should be avoided.
1. Allan C. Halpern, M.D. Chief of Dermatology at Memorial Sloan-Kettering Cancer Center
A nationally recognized dermatologist, Dr. Halpern has made significant contributions to predicting the frequency of the occurrence of skin cancer and dysplastic nevi (irregularly shaped moles that may become cancerous) in large populations. He is also an authority on early detection, tracking, and treatment of such skin changes to prevent them from progressing to advanced cancer. His work has led to the design of strategies for estimating a person’s risk of melanoma.
“Vitamin-D Research Turning Sunscreen Wisdom on Head”
Phoenix Arizona Republic Abstract, 22 May 2005
The thinking is that even if too much sun leads to the non-lethal skin cancer, too little sun may be worse. “I find the evidence that the sun prevents cancer to be mounting and increasingly compelling.” — Dr. Allan Halpern M.D., Dermatology Chief
Comment: The Chief of Dermatology at Memorial Sloan-Kettering has seen the scientific research and has the courage to admit that our past opinions about the sun appear to be wrong.
2. Samuel S. Epstein, M.D., author of The Politics of Cancer Revisited, is one of the foremost environmental toxicologists in the world. Epstein and his supporters point out that chemical sunscreens often contain toxic and even carcinogenic chemicals, proven to cause DNA mutation in laboratory studies — though it is still debatable whether they behave this way in human skin. Such chemicals include benzophenones, 4-MBC, placenta (and other estrogenic compounds), talc (a mineral similar to asbestos) and even that old stand-by PABA, which, though a nutrient in the B-vitamin family, produces potentially dangerous substances when activated by UV rays.
International Leading Authority on the Causes and Prevention of Cancer
Samuel S. Epstein, M.D. is professor emeritus of Environmental and Occupational Medicine at the University of Illinois School of Public Health, and Chairman of the Cancer Prevention Coalition. He has published some 260 peer reviewed articles, and authored or co-authored 11 books including: the prize-winning 1978 The Politics of Cancer; the 1995 Safe Shopper’s Bible; the 1998 Breast Cancer Prevention Program; the 1998 The Politics of Cancer, Revisited; the 2001 GOT (Genetically Engineered) MILK! The Monsanto rBGH/BST Milk Wars Handbook; the 2001 Unreasonable Risk. How to Avoid Cancer from Cosmetics and Personal Care Products: The Neways Story; and the 2005 Cancer-Gate: How to Win the Losing Cancer War.
Dr. Epstein is an internationally recognized authority on avoidable causes of cancer, particularly unknowing exposures to industrial carcinogens in air, water, the workplace, and consumer products–food, cosmetics and toiletries, and household products including pesticides–besides carcinogenic prescription drugs.
Biography http://www.preventcancer.com/about/epstein.htm#bio
Presentations and Events http://www.preventcancer.com/about/epstein.htm#present
Awards http://www.preventcancer.com/about/epstein.htm#honors
Scientific Publications http://www.preventcancer.com/about/epstein.htm#sci
3. In 1998, Dr. Marianne Berwick, Epidemiologist at Memorial Sloan Kettering Cancer Center, reported at a meeting that her analysis of 16 epidemiology studies suggested that the more sunscreen a person used the higher chance of developing skin cancer.
4. Dr. Frank Garland, of the University of California, believes the increased use of chemical sunscreens is the primary cause of the epidemic of skin cancer.
5. Gordon Ainsleigh, D.C. believes that the use of sunscreens causes more cancer deaths than it prevents. He estimated that the 17% increase in breast cancer observed between 1991 and 1992 may be the result of the pervasive use of sunscreens over the past decade. (Preventive Medicine, Vol. 22, Feb. 1993)
6. Professor John Moan, of the Norwegian Cancer Institute, found the yearly increase in melanoma in Norway had increased 350% for men and 440% for women from 1957-1984, when sunscreens were first introduced.
7. Drs. Cedric and Frank Garland, of the University of California, have pointed out that while sunscreens do prevent against sunburn there is no scientific proof they protect against basal cell carcinoma in humans.
8. Sunlight, Nutrition and Health Research Center (SUNARC) Organization is devoted to research and education relating to the prevention of chronic disease through changes in diet and lifestyle.
Scientific advisors to SUNARC:
Greg A. Plotnikoff, M.D.
Department of Internal Medicine
University of Minnesota Medical School
Minneapolis, Minn USA
George P. Studzinski, M.D., Ph.D. Professor of Pathology & Laboratory Medicine
UMD-New Jersey Medical School
185 South Orange Ave
Newark, NJ 07103 USA
John Jacob Cannell, MD
The Vitamin D Council
Atascadero, CA
Health Concerns and Facts about Sun Exposure
The increasing occurrence of skin cancer in the USA is a major concern of physicians. New research shows that the active ingredients (toxic chemicals) in some sunscreens may contribute to genetic damage and skin cancer (The Scientist, Mar/Apr. 1999: 7). In fact, the sunscreen chemicals can absorb the rays’ energy, become energized, and chemically reactivate themselves, which can lead to chemical reaction in the skin that creates free radicals that cause DNA damage.
Toxic Ingredients to Avoid
Benzophenones (dixoybenzone, oxybenzone)
PABA and PABA esters (ethyl dihydroxy propyl PAB, glyceryl PABA,
p-aminobenzoic acid padimate O or Octyl dimethyl PABA)
Cinnamates (cinoxate, ethylhexyl, p-methoxycinnamate)
Salicylates (ethylhexyl salicylate, homosalate, octyl salicylate)
Avobenzone (butyl-methyoxydibenzoylmethane; Parsol 1789)
Digalloyl trioleate
Menthyl anthranilate
Propylene Glycol: Called a humectant in cosmetics, is industrial antifreeze, and the major ingredient in brake and hydraulic fluid. Material data sheets on Propylene Glycol warn to avoid skin contact as it is systemic and can cause liver abnormalities and kidney damage. If antifreeze is spilled on your garage floor and a dog or cat has one lick, it will kill them.
Chemical Composition of Sunscreens
Chemical sunscreens contain 2-5% chemical compounds. Benzophenone and similar compounds are some of the most powerful free radical generators known to man. They are used in industrial processes as free radicals to initiate chemical reactions. When activated by ultra violet light, the molecule doubles the bond to produce two free radical sites. The free radicals then initiate a chain reaction that can increase skin damage and increase skin cancer.
Worldwide the greatest risk in melanoma has been in countries where sunscreens have been heavily promoted. Queensland, Australia, where the medical establishment has vigorously promoted the use of sunscreens has more evidence of melanoma per capita than any other place. (Garland, Cedric F., et al. American Journal of Public Health, April 1992).
In 1997, Europe, Canada and Australia limited the formula for sunscreens to three specific ingredients: avobenzone, titanium dioxide and zinc oxide. All others were banned.
Best of health to you
Answer by quijibored
No, not true, regardless of sun exposure or vitamin D intake. People with basal or squamous cell skin cancers have a higher rate of second malignancies than people who have never had a non-melanoma skin cancer.
Cancer Causes Control. 2000 Dec;11(10):891-7.
Association of basal cell skin cancers with other cancers (United States).Friedman GD, Tekawa IS.
Division of Research, Kaiser Permanente Medical Care Program, Oakland, California, USA. [email protected]
BACKGROUND: Persons with basal cell skin cancer (BCSC) have shown increased risk of developing cancer at several other sites. METHODS: We identified 3164 persons with BCSC and 15,730 comparison subjects matched for age, sex, race, residence area and length of membership in a health maintenance organization. RESULTS: In retrospective follow-up for up to 24 years (mean 11.3 years), BCSC patients experienced statistically significant increases in the incidence of all cancer (relative risk [RR] = 1.2, 95% confidence interval [CI] = 1.1-1.4) lung cancer (RR = 1.4, CI = 1.0-1.8) and melanoma (RR = 2.2, CI = 1.6-3.0). Women experienced significantly increased risk for all cancer, lung cancer, melanoma and thyroid cancer, increases of borderline significance in breast cancer, non-Hodgkin’s lymphoma and leukemia, and increased pre-existing bladder cancer. Men showed statistically significant increases in all cancer, melanoma, and kidney cancers, and mouth and throat cancers. Multivariate analysis incorporating available risk factor data did not weaken positive associations with BCSC except slightly for melanoma and for bladder cancer in women. Other previously reported associations were not confirmed. CONCLUSION: Periodic skin examinations appear well justified after removal of BCSC to detect new skin cancers including melanoma. Given the relatively weak, unexplained associations of BCSC with internal cancers, the costs vs. benefits of extra efforts to detect the latter still need to be determined.
PMID: 11142523
Risk of subsequent cancer following invasive or in situ squamous cell skin cancer.Efird JT, Friedman GD, Habel L, Tekawa IS, Nelson LM.
Division of Epidemiology, Department of Health Research and Policy, Stanford School of Medicine, Stanford, CA 94305-5405, USA.
PURPOSE: Determine the risk of subsequent cancer following squamous cell skin cancer. METHODS: Using computerized surgical pathology records and membership data from a health maintenance organization, we retrospectively identified 822 individuals with primary squamous cell skin cancer (SCSC) and 3662 comparison subjects matched for age, sex, race, residence area, and length of membership. Patients were included in the study if they had no prior history of cancer, and received at least one multiphasic health checkup and questionnaire (MHC). Patients were followed for subsequent invasive cancer up to 24 years, with a mean follow-up time of 7.8 years. RESULTS: SCSC patients had a significantly greater risk [adjusted for body mass index (BMI) and education] for subsequent cancer overall (excluding non-melanoma skin cancer) [risk ratio (RR) = 1.4, 95% confidence interval (CI) = 1.2-1.6], and for basal cell skin cancer (RR = 13.8, 95% CI = 8.8-21.9), digestive (RR = 1.6, 95% CI = 1.1-2.4), and genitourinary cancers (RR = 1.5, 95% CI = 1.0-2.0). An increased, but not statistically significant, adjusted risk (RR > or = 1.4) was also observed for lip, oral cavity, and pharynx cancer (RR = 3.9, 95% CI = 0.6-25.0); non-cutaneous squamous cell cancer (RR = 1.9, 95% CI = 0.9-4.4); and respiratory and intrathoracic cancer (RR = 1.4, 95% CI = 0.8-2.6). The addition of alcohol consumption, combined occupational exposure, marital status, and smoking history to the multivariate model did not materially change any significant positive associations with SCSC. CONCLUSIONS: Our results suggest that patients diagnosed with SCSC may be at an increased risk of subsequent cancer at many sites, although several estimated risk estimates were within the limits of chance given no true association.
PMID: 12377424
Also, mercola.com is not an unbiased source of health information, hence the .com address. The info posted there is slanted towards alternative medicine.
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